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CATECHIN
1.5oz/$13.99
Catechin
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Primarily for those drinking green tea to heal through its active ingredient: Catechin. Tannin in green tea is mostly catechin and forms a key component in its astringent taste. NOTE: Individuals with allergies to catechin-rich foods like red wine, dark chocolate (cocoa), cherries, apples & cranberries should consult a physician before drinking. This is a concentrated fresh catechin product.

OI OCHA KOIAJI
3.5oz/$17.99
Koiaji Sencha
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This special Sencha tea has an extra 300 miligram shot of the natural active ingredient in green tea, Catechin, added to it.

Green Tea: Scientific Fact - Green tea has numerous benefits for disease prevention and anti-aging purposes. Here we review how green tea protects many aspects of our physiology.

Green tea research has shown it has an effect on variety of health conditions including heart health, hypertension, circulation, cholesterol, diabetes, the brain, strokes, the liver, the kidneys, the bowels, the skin, and its antioxidant, anti-inflammatory, anti-viral and anti-bacterial qualities. The ability to lower blood sugar, chelate iron and control the production of nitric oxide are all especially important. This ancient beverage seems custom-made to protect health and delay aging.

Cardio-Protective Properties of Green Tea - Blood sugar tends to increase with age, accelerating aging by cross-linking with proteins (glycation). Green tea can lower serum glucose levels, an important anti-aging benefit.
A study comparing the effects of 75-day feeding of green tea and black tea to aged rats found that green tea lowered blood sugar only slightly better than black tea (23.9% vs 22.8%), but was markedly superior in reducing triglycerides (33.3% vs 25%; high triglycerides are strongly associated a high risk of cardiovascular disease). A low ratio of triglycerides to HDL is an excellent marker of cardiovascular health.

Black tea, however, was a better inducer of superoxide dismutase (SOD; the activity of SOD was 117% higher in the black tea group vs. control, as compared to 90.8% higher in the green tea group), and a better blocker of the harmful malondialdehyde, a byproduct of lipid peroxidation (black tea reduced it by 34.6%; green tea by 25.4%). The authors' conclusion that black tea is a more powerful antioxidant in vivo needs to be confirmed by other studies.

Population-based studies indicate that the antioxidant properties of green tea may help prevent atherosclerosis, particularly coronary artery disease. (Population-based studies means studies that follow large groups of people over time and/or studies that are comparing groups of people living in different cultures or with different dietary habits, etc.) In May 2006, however, the U.S. Food and Drug Administration (FDA) rejected a petition from teamakers to allow tea labels to claim that green tea reduces the risk of heart disease. The FDA concluded that there is no credible evidence to support qualified health claims for green tea or green tea extract reducing the risk of heart disease.
Hypertention Protective Properties of Green Tea - Tea possesses hypotensive effects in Chinese medicine. Conflicting results in human and animal trials are demonstrated between tea consumption and blood pressure.
Epidemiological evidence about the long-term effect of tea on hypertensive risk is also inconsistent. The Department of Family Medicine, Medical College, National Cheng Kung University, Taiwan examined the effect of tea drinking, measured in detail for the past decades, on the risk of newly diagnosed hypertension in 1507 subjects (711 men and 796 women), 20 years or older, who did not have a hypertensive history during 1996 in Taiwan. RESULTS: Six hundred subjects (39.8%) were habitual tea drinkers, defined by tea consumption of 120 mL/d or more for at least 1 year. Compared with nonhabitual tea drinkers, the risk of developing hypertension decreased by 46% for those who drank 120 to 599 mL/d and was further reduced by 65% for those who drank 600 mL/d or more after carefully adjusting for age, sex, socioeconomic status, family history of hypertension, body mass index, waist-hip ratio, lifestyle factors (total physical activity, high sodium intake, cigarette smoking, alcohol consumption, and coffee drinking), and dietary factors (vegetable, fruit, unrefined grain, fish, milk, visible-fat food, and deep fried food intake). However, tea consumption for more than 1 year was not associated with a further reduction of hypertension risk. CONCLUSION: Habitual moderate strength green or oolong tea consumption, 120 mL/d or more for 1 year, significantly reduces the risk of developing hypertension in the Chinese population.

Green Tea & Circulation - The vasodilating effects of tea are documented. One study compared the effect of coffee, tea, hot water with caffeine, and plain hot water on skin temperature, indicating peripheral vasodilation.
Tea produced the greatest vasodilating response. The authors speculate that this is due to the action of catechins. An increase in peripheral circulation is valuable for oxygenating tissue, and is also associated with a relaxed mood. Hence some alternative experts have advised drinking green tea in the evening as a relaxant.

A more detailed recent study compared the effectiveness of various catechins as vasorelaxants in rat arteries. All four main catechins present in green tea were shown to have a dose-dependent vasodilating effect, with epigallocatechin gallate being the most potent. Like human estrogens, catechins may act as calcium-channel blockers. Vasodilation is one of the cardioprotective effects of estrogens. Thus, green tea extract might be of particular importance to estrogen-deficient postmenopausal women.

Green tea catechins containing the galloyl group (epigallocatechin gallate, epigallocatechin, and epicatechin gallate) have been found to inhibit the proliferation of smooth muscle cells lining blood vessels in vitro (estrogens and progesterone also show this antiproliferative action; hence the natural protection against atherosclerosis seen in premenopausal women). Smooth muscle proliferation is one of the crucial processes involved in atherosclerosis and heart disease. One mechanism of the antiproliferative action of catechins is apparently the inhibition of protein tyrosine kinase activity (which is also involved in tumor growth).

The authors conclude that "tea catechins may be useful as a template for the development of drugs to prevent the pathological changes of atherosclerosis and post-angioplasty restenosis." (Restenosis is the narrowing of blood vessels after surgery, usually due to the rapid regrowth of plaque.) It seems more logical to use green tea for prevention of atherosclerosis to start with. Green tea lowers fibrinogen, and inhibits excessive clotting and platelet aggregation.

A recent American in vivo study using hamsters found that while both green tea and black tea improved plasma lipid profiles and protected cholesterol against oxidation, green tea also lowered fibrinogen significantly more than black tea. One of the green tea polyphenols, epicatechin, was found to be able to significantly inhibit the production of thromboxane, one of the compounds required for platelet aggregation.


Cholesterol Lowering Properties of Green Tea - Research shows that green tea lowers total cholesterol and raises HDL ("good") cholesterol in both animals and people. When rats were fed 2.5% green tea leaves in their
diet the experimental group showed a drop in total cholesterol, low-density cholesterol, & triglycerides. The body weight of green tea-fed rats was 10 to 18% lower than that of rats not consuming green tea. In addition, the activity of antioxidant enzymes superoxide dismutase (SOD) and catalase, and of anti-carcinogenic phase-II enzyme glutathione S-transferase (GST), were significantly higher in the green tea group, as was the glutathione level in the liver. There was no liver or kidney toxicity. Thus, the study demonstrated combined cardiovascular and anticancer effects of green tea.

The cholesterol-lowering (hypocholesterolemic) effects of green tea (as well as black tea) have been confirmed by both animal and human epidemiological studies. In addition to lowering the atherogenic index as expressed by the HDL/total cholesterol ratio in rats, green tea and jasmine green tea also reduced the increase of liver weight that results from fat deposition. High consumption of green tea by humans, especially more than 10 cups a day, was found to be associated with higher HDLs and lower LDL and VLDL cholesterol, as well as with various bio-markers indicating better liver health. Lower levels of lipid peroxides in the liver are one well-confirmed benefit of green tea supplementation, found in study after study. The pancreas is another organ that is protected by green tea.

One population-based study found that men who drink green tea are more likely to have lower total cholesterol than those who do not drink green tea. Results from one animal study suggest that polyphenols in green tea may block the intestinal absorption of cholesterol and promote its excretion from the body. In another small study of male smokers, researchers found that green tea significantly reduced blood levels of harmful LDL cholesterol.

In a double-blind, randomized, placebo controlled trial done by Division of Cardiovascular Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, 240 adults were given either theaflavin-enriched green tea extract in form of 375mg capsule daily or a placebo. After 12 weeks, patients in the tea extract group have significantly less low-density lipoprotein cholesterol (LDL-C) and total cholesterol (16.4% and 11.3% lower than baseline, p<0.01) then the placebo group. The author concluded that theaflavin-enriched green tea extract can be used together other dietary approaches to reduce LDL-C.

Green tea may also lower intestinal fat absorption. One animal study found that rats fed a diet containing a significant amount of catechin had a higher excretion of fat in the feces compared to the control group on a polyphenol-free diet. If this holds for humans who take the green tea extract, then it's good weight-loss and cardiovascular news.

Supplementation with antioxidants is important in part because by protecting cholesterol from oxidation, antioxidants help protect against atherosclerosis. In an animal study comparing the effectiveness of various antioxidants in preventing the oxidation of VDL and LDL cholesterol, vitamin E, genistein (phytoestrogen found chiefly in soy products) and green tea were found to be effective antioxidants, with genistein being particularly effective (oxidation lag time of 49% on the high-genistein diet), but green tea also exerting considerable activity (lag time of 33%). It would be interesting to see the results of combined genistein and green tea supplementation, particularly in humans. On the other hand, it could be argued that this is precisely the case of the Japanese diet. Japan enjoys the longest life expectancy in the world, and the lowest cardiovascular mortality for men, in spite of heavy smoking.


Anti-Diabetic Properties of Green Tea - The ability to significantly lower blood glucose has been confirmed also in studies using diabetic rats. Both green and black tea possess anti-diabetic activity, and are effective in the
prevention and treatment of diabetes. The fact that aged rats responded so dramatically to these polyphenols implies that it is possible to reverse the age-related rise in glucose intolerance and the resulting degenerative cascade of atherosclerosis and other degenerative disorders.

In what way are tea polyphenols able to lower serum glucose? The main mechanism seems to be the inhibition of the activity of starch digesting enzyme amylase. Tea inhibits both salivary and intestinal amylase, so that starch is broken down more slowly, and the rise in serum glucose is thus minimized. In addition, according to one recent study, tea may reduce the intestinal absorption of glucose.

A relatively little known compound found in onions and in tea, especially green tea, called diphenylamine, seems to have a strong sugar-lowering action. Again, the lesson here is that we are barely beginning to identify the significant phenolic compounds and their interactions; it's best not to rely on a single ingredient such as epigallocatechin gallate, but rather to ingest the whole complex set of bioactive compounds present in tea for best results.

Thanks to the serum glucose-lowering effect of tea, we thus obtain significant anti-aging benefits of calorie restriction, reduced glycation, and lower insulin secretion. If you drink tea with a carbohydrate-rich meal, you slow down the release of glucose and reduce its absorption (you also reduce the absorption of iron, another anti-aging benefit). Thus, you prevent the harmful spiking of insulin. Since insulin is our most fattening hormone and, with cortisol, our most pro-aging hormone, you also derive the substantial range of benefits that go with calorie restriction and insulin control.

Green tea has been used traditionally to control blood sugar in the body. Animal studies suggest that green tea may help prevent the development of type 1 diabetes and slow the progression once it has developed. People with type 1 diabetes produce little or no insulin, a hormone that converts glucose (sugar), starches, and other foods into energy needed for daily life. Green tea may help regulate glucose in the body.



Green Tea & Brain Functioning - Green tea has been shown to protect the brain from oxidative stress, and lower monoamine oxidase (MAO) activity. Neurodegenerative diseases have been linked both to free radical damage and to excessive
breakdown of neurotransmitters caused by high monoamine oxidase activity. Green tea in general, as well as its phenolic components catechin and epigallocatechin gallate, have been found to be effective at inhibiting MAO and lowering peroxide levels in glial cells in the brain.

Further evidence that green tea might be useful in preventing age-related brain degeneration comes from studies of the effects of catechins on nerve cell cultures. Thanks to their antioxidant properties, catechins were able to protect the cells from death induced by glucose oxidase. Catechins are also able to restrain the production of nitric oxide by the glial cells surrounding the neurons. Nitric oxide plays an important role as a neurotransmitter involved in memory formation, but excess levels lead to neural death and neurodegenerative disorders. Flavonoids in general are particularly effective in regulating the levels of nitric oxide in the brain. Green tea is among a handful of substances that can reduce nitric oxide production at concentrations of less than 300 parts per million.

The ability to lower lipid peroxidation and to chelate iron in the nervous system is also of tremendous importance. Parkinson's disease involves the progressive destruction of the dopamine-releasing nigrostriatal dopaminergic system, and hence the ever-increasing dopamine deficiency. It is possible that sufficient green tea consumption might protect against Parkinson's disease. Both green and black tea are also potent inhibitors of intestinal absorption of non-heme iron, lowering the amount of free iron reaching the brain in the first place.

Japan has a much lower rate of Alzheimer's disease than Western countries. The Japanese living in Hawaii have 2.5 times the incidence of Alzheimer's disease than do native Japanese in Japan. It would be interesting to see to what extent the Japanese custom of sipping green tea all day, as contrasted with much lower consumption of green tea by the American-Japanese, contributes to neural protection.

Researchers including Edward Okello of Newcastle University (in northeast England) biology school pitted green tea, black tea, and coffee. They found that tea stops the activity of chemicals in the brain associated with the development of Alzheimer's disease. Symptoms of Alzheimer's disease occur when brain nerve cells that process information and memory degenerate and die. Abnormalities such as plaque and tangles proteins form on nerve cells.

The first brain chemical, acetylcholinesterase (AchE), breaks down one of the brain's chemical messengers that helps transport and process information -- called acetylcholine. A drop in acetylcholine, in areas of the brain associated with memory and learning, has been linked to Alzheimer's disease. Medications used to treat Alzheimer's disease work by inhibiting acetylcholinesterase. Aricept, Exelon, and Reminyl are examples of these medications.

The teas also stopped the activity of other chemicals known to be key in making plaques and tangles in the brains of Alzheimer's patients. The second chemical is called butyrylcholinesterase (BuChE). However, green tea also inhibited a third and final brain chemical called beta-secretase, which is also involved in brain protein deposits seen with Alzheimer's disease.

There is also some evidence that iron plays a part in epilepsy. Green tea polyphenols have been found to inhibit or diminish iron-induced epileptic seizures, and to inhibit the hyperactivity of dopaminergic neurons. It is in fact likely that green tea, especially the decaffeinated kind, acts as a mild sedative.
Theanine, an amino acid found in green tea, has also been found to have beneficial effects by raising the levels of serotonin and/or dopamine in various important brain regions, particularly the hypothalamus, hippocampus (memory center), and striatum.


Green Tea & Strokes - A Japanese study of almost 6000 nonsmoking women over the age of 40 showed that those who drank five or more cups of green tea a day had only half the incidence of stroke compared with women who
drank less than five cups. A smaller Dutch study found an even more dramatic effect in men who drank a lot of black tea: those men who drank close to five cups of tea a day had only 31% risk of stroke compared to those who drank less than about two and a half cups of tea. These results were not replicated in England, however; based on existing in vivo plasma antioxidant measurements, it has been suggested that the English custom of putting cream or milk in tea destroys all antioxidant benefits.

A Japanese animal study done on spontaneously hypertensive rats has confirmed that epigallocatechin gallate reduces the incidence of stroke and prolongs life span.
Green Tea & The Liver - Population-based studies have shown that men who drink more than 10 cups of green tea per day are less likely to develop disorders of the liver. Green tea also seems to protect the liver from the
damaging effects of toxic substances such as alcohol.

Results from several animal and human studies suggest that one of the polyphenols present in green tea, known as catechin, may help treat viral hepatitis (inflammation of the liver from a virus). In these studies, catechin was isolated from green tea and used in very high concentrations. It is not clear whether green tea (which contains a lower concentration of catechins) confers these same benefits to people with hepatitis.

Green Tea & Kidney Health - The kidneys are another area where green tea has shown to have protective effects. Decreased kidney function due to aging and kidney failure are a frequent cause of death.

The public is generally unaware that anything can be done to prevent the age-related decline in kidney function. Making use of a wide-range of antioxidant protection appears crucial, and flavonoids, including green tea catechins, are very potent antioxidants. Epigallocatechin gallate was shown to induce antioxidant enzymes in the kidneys, as well as to reduce uremic toxins in the blood, suggesting improved kidney function in an animal model of kidney failure.Kidney problems are often associated with high blood sugar and consequent glycosylation of various proteins (hence the strong link between kidney failure and diabetes). Since green tea has the ability to lower serum glucose, this is another way in which it helps protect against kidney failure. Likewise, the antioxidant properties of green tea likely play a significant role in protecting the kidneys. Since green tea has been shown to lower the concentrations of free radicals and lipid peroxides in organs such as the liver and the pancreas, this is likely to be true in the kidneys as well.

Maintaining kidney health is a crucial though often neglected part of anti-aging medicine. Green tea is one of the resources we have for protecting this critical detoxification system, and it seems to be a particularly powerful one.
Green Tea & Inflammatory Bowel Disease (IBD) - Green tea may help reduce inflammation associated with Crohn's disease and ulcerative colitis, the two types of IBD.

If green tea proves to be helpful for preventing colon cancer, this would be an added benefit for those with IBD because they are at risk for colon cancer.

Green Tea & Wrinkles - Coffee raises levels of cortisol and insulin hormones that accelerate aging and store body fat. Green tea contains catechin polyphenols, antioxidants that boost metabolism and slow aging.

Green tea also blocks absorption of bad fats by 30 percent, while theonine promotes calmness and improves mood.

Initial experiments show that green tea's antioxidant properties help to prevent sunburn, wrinkles, and even skin cancer.Although green tea extracts have shown to help prevent and skin diseases in mice, research has not yet been done with humans. But it is cautioned that green tea was a preventive step, not a cure, for skin cancer. And it is not clear if applying tea to the skin is the same as drinking it. This, however, did not stop green tea from being used in more and more skin products, including lotions, creams, bath and shower wash, and shampoo.
Antioxidant Properties of Green Tea - Green tea provides a wealth of simple phenolics; black tea provides more complex polyphenols. There is some controversy about which group has higher antioxidant potential in vivo.

An Italian study found that while green tea was six times more potent in inhibiting lipid peroxidation in vitro, when healthy human subjects ingested the same amount of either black or green tea, the plasma antioxidant capacity (expressed as TRAP, or total radical-trapping antioxidant parameter) was similar in both groups.

The increase in plasma antioxidant capacity was quite rapid, peaking at 30-50 minutes. This indicates that the most bioactive antioxidant compounds are quickly absorbed in the upper part of the gastrointestinal system.

Interestingly, while the addition of milk did not diminish antioxidant activity of either black or green tea in vitro, it completely abolished it in vivo, according to an Italian study. Some authors, however, think that skim milk does not significantly interfere with the absorption of polyphenols.

One study found green tea polyphenols to be comparable to BHT (a well-known synthetic phenolic antioxidant) in antioxidant power when it came to protecting canola oil; green tea outperformed BHT when the oil was heated.

Catechins have also been found to outperform Vitamin C and beta-carotene ten times in scavenging the alkyl peroxyl radical. One study found green tea polyphenols to be more potent antioxidants than Vitamin C, Vitamin E, rosemary extract, and even curcumin in some systems.

Diabetics show premature aging and poor immune function due to the high oxidative stress that results from high serum glucose. When type-II diabetics were put on a diet that included 6 cups of tea a day plus some tomato sauce with onions, oxidative damage to the DNA of their lymphocytes was found to be markedly reduced. We already know that carotenoids can protect DNA; it is possible that compounds found in green tea may also be able to protect DNA, especially in synergistic action with other antioxidants.

One of the antioxidant properties of green tea catechins is their ability to protect against the ravages of oxidized linoleic acid - an omega-6 fatty acid usually consumed in excess in the Western world, from sources such as margarine, safflower oil and corn oil; excess linoleic acid (most likely in the peroxidized form, which activates various pro-inflammatory enzymes) is involved in promoting inflammation, tumor growth, and most degenerative disorders. Few people are aware that when polyunsaturated cooking oils are heated, the result is carcinogenic peroxides. A recent study discovered that catechins synergize with alpha-tocopherol (Vitamin E) to protect against oxidative damage by oxidized linoleic acid before this fatty acid is incorporated into cell membranes.

Catechins also have a sparing effect on Vitamin E (alpha-tocopherol) and beta-carotene, resulting in overall greater antioxidant protection of the polyunsaturated fatty acids that are incorporated into cell membranes. The sparing effect on tocopherol may be due to the fact that catechins are effective scavengers of aqueous oxygen radicals, and thus prevent their entry into the lipids, where these radicals would oxidize alpha-tocopherol. It is also possible that catechins can regenerate the antioxidant power of alpha-tocopherol by donating a hydrogen molecule to its oxidized form. Thus it appears that green tea polyphenols, like flavonoids in general, boost the levels of other antioxidants.

Of special interest is the ability of catechins to inhibit the dangerous peroxynitrite free radical, a strong reactant oxidant formed when the potent free radical superoxide encounters nitrogen-containing compounds such as nitric oxide. Peroxynitrite destroys proteins, as well as vital antioxidants such as glutathione and Vitamin E. Green tea catechins were shown to be more effective than Trolox, a synthetic antioxidant, in protecting critical amino acids such as tyrosine and the amino acids of apolipoprotein B in LDL cholesterol against nitration damage by the peroxynitrite radical.

Another interesting recent discovery concerns one particular green tea polyphenol called epicatechin. This particular polyphenol appears to be metabolized to an anthocyanin-like compound that is also an antioxidant, offering particularly long-lasting protection.
Returning to the question of antioxidant properties of simple catechins in green tea versus the complex polymeric polyphenols in black tea, a new study found that in lipids the simple compounds were more effective, while in aqueous conditions polymers had more activity, but only up to a point. Glycosylation, meaning cross-linking with glucose, decreased the antioxidant properties of all compounds tested.
Anti-Inflammatory Properties of Green Tea - Green tea polyphenols are also effective anti-inflammatory agents. Chronic inflammation has been linked to serious degenerative disorders associated with aging,

including cardiovascular disease, cancer, and Alzheimer's disease. An important part of the inflammatory process is the excess production of nitric oxide, which in turn leads to the production of carcinogenic nitrous compounds. Epigallocatechin gallate has been found to inhibit the enzyme nitric oxide synthase, thus reducing the production of inflammation-related NO.

Tumor necrosis factor alpha also plays a pivotal part in inflammation. It has been found that green tea polyphenols downregulate the gene that causes the release of tumor necrosis factor alpha. Thus, it can be concluded that green tea reduces the inflammatory response by controlling the production of nitric oxide and tumor necrosis factor alpha.

Antioxidants in green tea may prevent and reduce the severity of rheumatoid arthritis, according to a study from CWRU's School of Medicine study published in the April 13 issue of the Proceedings of the National Academy of Sciences.

The study examined the effects of green tea polyphenols on collagen-induced arthritis in mice, which is similar to rheumatoid arthritis in humans. Polyphenols are chemicalsthat occur naturally in certain foods, including green tea, and many work as antioxidants to protect the bodyfrom oxidative stress that causes disease.

In each of three different study groups, the mice given the green tea polyphenols were significantly less likely to develop arthritis. Of the 18 mice that received the green tea, only eight (44 percent) developed arthritis. Among the 18 mice that did not receive the green tea, all but one (94 percent) developed arthritis. In addition, researchers noted that the eight arthritic mice that received the green tea polyphenols developed less severe forms of arthritis. Although there is no epidemiologic data, anecdotal evidence indicates that people in countries where green tea is consumed are far less likely to have rheumatoid arthritis, said the study's senior author, Hasan Mukhtar, professor of dermatology.
Virus Replication & Green Tea - Recent studies suggest that green tea catechins may inhibit the HIV virus replication, and various other viruses. A study done at the Laboratory of Viral Oncology in Nagoya, Japan, discovered that
two catechins found in green tea, epigallocatechin gallate and epicatechin gallate, were able to differentially inhibit the enzymes used by the HIV virus for replication: reverse transcriptase and various DNA and RNA polymerases. A more recent Chinese study at the Institute of Medical Biotechnology in Beijing found that green tea catechins in general could inhibit the reverse transcriptase or polymerases of several types of viruses, including HIV-1 and herpes simplex 1. Various polymeric oxidation products of polyphenols have also been found to inhibit the herpes simplex virus. It seems that flavonoids in general ought to be more thoroughly researched for their ability to inhibit the replication of viruses and keep them in a state of latency.

Anti-Bacterial Properties of Green Tea - A discovery related to the antibacterial properties of green tea polyphenols found that these compounds inhibit the growth and adherence of oral bacteria.
Green tea extract has been found to strongly inhibit periodontal-causing bacterium, Porphyromonas, and decay-causing bacteria such as Streptococcus salivarius and Streptococcus mutans. A Chinese study showed that Streptococcus mutans could be inhibited completely by sufficient contact with green tea polyphenols. Using green tea as a mouth rinse resulted in less plaque and periodontal disease. Black tea has also been found effective. One possible mechanism of the action of tea in preventing dental decay is its ability to inhibit the enzyme amylase present in the saliva. Thus, less starch gets converted in the mouth into bacteria-feeding simple sugars such as glucose and maltose. Bacterial amylase is likewise inhibited, making less nutrition available to the decay-causing organisms.

Green tea catechins also help destroy harmful intestinal bacteria. When tube-fed patients received 300 mg of tea catechins a day, the putrefactive products in their gastrointestinal tract decreased, and organic acids increased, lowering the pH. The greater acidity is highly beneficial, since it makes the environment inhospitable to harmful bacteria, while beneficial lactic acid bacteria can thrive. Indeed, the bactericidal activity of green tea does not affect lactic acid bacteria. Decreased levels of putrefactive products and improved intestinal flora lead to better digestion, better immune function, and lower risk of colorectal cancer.
References - Life Extension Magazine June 1999 and other studies added.
Alic M. Green tea for remission maintenance in Crohn's disease? Am J Gastroenterol. 1999;94(6):1710.

Anderson JW et al. Selective effects of different antioxidants on oxidation of lipoproteins from rats. Proc Soc Exp Biol Med 1998;218:376-81.

Archives of Dermatology, 2000;136:989-994, 1051.

Bravo L, Abia R et al. Degradation of polyphenols (catechin and tannic acid) in the rat intestinal tract. Effect on colonic fermentation and fecal output. Br J Nutr 1994;71:933-46.

Chan MM et al. Inhibition of inducible nitric oxide synthese gene expression and enzyme activity by epigallocatechin gallate, a natural product from green tea. Biochem Pharmacol 1997; 54:1281-86.

Chen ZY, Chan PT. Antioxidant activity of green tea catechins in canola oil. Chem Phys Lipids 1996; 82:163-72.

Chen PC, et al. A green tea-derived polypheol, epigallocatechin-3-gallate, inhibits IkappaB kinase activation and IL-8 gene expression in respiratory epithelium. Inflammation 2002;26:233-41

Choi JH et al. Effects of green tea catechins on hepatic microsomal phospholipase. J Nutr Sci Vitaminol 1998; 44:673-83.

Chung HY et al. Peroxynitrite-scavenging activity of green tea tannin. J Agric Food Chem 1998; 46:4484-86.

Deng ZY, Tao BY, et al. Effect of green tea and black tea on blood glucose, triglycerides, and antioxidants in aged rats. J Agricult Food Chem 1998;46:3875-78.

Dulloo AG, et al. Efficacy of a green tea extract rich in catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat oxidation in humans. Am J Clin Nutr 1999;70:1040

Gomes A et al. Anti-hyperglycemic effect of black tea (Camellia sinensis) in rat. J Ethnopharmacol 1995; 45: 223-26.

Graham HN. Green tea composition, consumption, and polyphenol chemistry. Prev Med 1992;21:334-50

Guo Q et al. Studies on protective mechanisms of four components of green tea polyphenols against lipid peroxidation in synaptosomes. Biochim Biophys Acta 1996; 1304:210-22.

Haqqi, Tariq M., Antioxidants in green tea may prevent and reduce the severity of rheumatoid arthritis, CWRU's School of Medicine study published in the April 13 issue of the Proceedings of the National

Hara Y., Academy of Sciences.Influence of tea catechins on the digestive tract. J Cel Biochem 1997; Suppl 27: 52-58.

Hayashi M et al. Effects of green tea extract on galactosamine-induced hepatic injury in rats. Nippon Yakurigaku Zasshi 1992; 100:391-99.

He YH, Kies C. Green and black tea consumption by humans: impact on polyphenol concentrations in feces, blood, and urine. Plant Foods Hum Nutr 1994; 46:221-9

Huang Y, Zhang AQ, et al. Vasorelaxant effects of purified green tea catechin derivatives in rat mesenteric artery. Life Sciences 1998;63:275-283.

Imai K, Nakachi K. Cross-sectional study of effects of drinking green tea on cardiovascular and liver diseases. BMJ 1995;310:693-96.
Kabuto H et al. Monoamine metabolites, iron induced seizures, and the anticonvulsant effects of tannins. Neurochem Res 1992; 6:585-90.

Kaneko T, Matsuo M, Baba N. Inhibition of linoleic acid hydroperoxide-induced toxicity in cultured human umbilical vein endothelial cells by catechins. Chem Biol Interact 1998; 114:109-19.

Karawya Ms. Et al. Diphenylamine, an antihyperglycemic agent from onion and tea. J Natural Prod 1984; 47:775-80.

Keli SO et al. Dietary flavonoids, antioxidant vitamins, and incidence of stroke: the Zutphen study. Arch Intern Med 1995; 156:637-42.

Kreydiyyeh SI et al. Tea extract inhibits intestinal absorption of glucose and sodium in rats. Comp Biochem Physiol C Pharmacol Toxicol Endocrinol 1994;108:359-65.

Lean ME et al. Dietary flavonols protect diabetic human lymphocytes against oxidative damage to DNA. Diabetes 1999; 48:176-81.

Lin AM et al. The antioxidant property of green tea against iron-induced oxidative stress in rat brain. Chin J Physiol 1998; 41:189-94.

Lin YL, Cheng CY, et al. Hypolipidemic effect of green tea leaves through induction of antioxidant and phase II enzymes including superoxide dismutase, catalase, and glutathione S-transferase in rats. J Agricult Food Chem 1998;46:1893-99.

Lin YL, Lin JK. Epigallocatechin gallate blocks the induction of nitric oxide synthase by downregulating lipopolysaccharide-induced activity of transcription factor nuclear factor-kappaB. Mol Pharmacol 1997; 52:465-72.

Lu LH, Lee SS, Huang HC. Epigallocatechin suppression of proliferation of vascular smooth muscle cells: correlation with c-jun and JNK. Brit J Pharmacol 1998;124:1227-37.

Luper S. A review of plants used in the treatment of liver disease: part two. Alt Med Rev . 1999;4(3):178-188.

Matsuoka Y et al. Ameliorative effects of tea catechins on active oxygen-related nerve cell injuries. J Pharmacol Exp Ther 1995; 274:602-8.

Maron D, et al. Cholesterol-lowering effect of a theaflavin-enriched green tea extract: a randomized controlled trial. Arch Intern Med. 2003 Jun 23;163(12):1448-53.

Mazzio EA et al. Food constituents attenuate monoamine oxidase activity and peroxide levels in C6 astrocyte cells. Planta Med 1998;64:603-6.

Nakane H, Ono K. Differential inhibition of HIV reverse transcriptase and various DNA and RNA polymerases by some catechin derivatives. Nucleic Acids Symp Ser 1989; (21): 115-16.

Nakao M, Takio S, Ono K. Alkyl peroxyl radical scavenging activity of catechins. Phytochemistry 1998;49:2379-82.

Okello, E. Phytotherapy Research, October 2004; vol 18: pp 624-627. News release, University of Newcastle upon Tyne.

Pannala AS et al. Inhibition of peroxynitrite-mediated tyrosine nitration by catechin polyphenols. Biochem Biophys Res Commun 1997; 232:164-68.

Parshad R, Sanford RR, et al. Protective action of plant polyphenols on radiation-induced chromatid breaks in cultured human cells. Anticancer Res 1998;18:3263-66.

Pricone, Nicholas Perricone, The Wrinkle Cure - The Perricone Prescription, Warner Books; (May 2001).

Pietta P, Simonetti P. Dietary flavonoids and interactions with endogenous antioxidants. Biochem Molec Biol International 1998;44:1069-74.

Plumb GW et al. Antioxidant properties of catechins and proanthocyanidins: effect of polymerization, galloylation and glycosylation. Free Radic Res 1999; 29:351-58.

Quinlan P, Lane J, Aspinall L. Effects of hot tea, coffee and water ingestion on physiological responses and mood: the role of caffeine, water, and beverage type. Psychopharmacology 1997; 134:164-73.

Rasheed A, Haider M. Antibacterial activity of Camellia sinensis extracts against dental caries. Arch Pharm Res 1998;21:348-52.

Sanaka S, Aizawa M, et al. Inhibitory effect of green tea polyphenols on growth and adherence of an oral bacterium, Porphyromonas gingivalis. Biosci Biotechnol Biochem 1996; 60:745-49.

Sato Y et al. Possible contribution of green tea drinking habits to the prevention of stroke. Tohoku J Exp Med 1989; 157:337-43.

Serafini M et al. In vivo antioxidant effect of green and black tea in man. Eur J Clin Nutr 1996;50:28-32.

Soliman KF, Mazzio EA. In vitro attenuation of nitric oxide production in C6 astrocyte cell culture by various dietary compounds. Proc Soc Exp Biol Med 1998; 218:390-97.

Takabayashi F, Harada N. Effects of green tea catechins on cerulein-induced acute pancreatitis in rats. Pancreas 1997; 14:276-79.

Tao P. The inhibitory effects of catechin derivatives on the activities of human immunodeficiency virus reverse transcriptase and DNA polymerases. Chung Kuo 1992; 14:334-38.

Taylor JR, Wilt VM. Probable antagonism of warfarin by green tea. Ann Pharmacother 1999;33:426-8.

Uchida S et al. Effect of epigallocatechin gallate on the life span of stroke-prone spontaneously hypertensive rats. Clin Exp Pharmacol Physiol 1995; Suppl 1: S302-3.

Vinson JA, Dabbagh YA. Tea phenols: antioxidant effectiveness of teas, tea components, tea fractions and their binding with lipoproteins. Nutr Res 1998; 18:1067-75.

Vinson JA, Dabbagh YA. Effect of green and black tea supplementation on lipids, lipid oxidation and fibrinogen in the hamster: mechanisms for the epidemiological benefits of tea drinking. FEBS Let 1998;433:44-46.

Wang H, Wu Y. Inhibitory effect of Chinese tea on N-nitrosation in vitro and in vivo. IARC Sci Publ 1991; 105:546-49.

Yang YC, et al.The protective effect of habitual tea consumption on hypertension. Arch Intern Med. 2004 Jul 26;164(14):1534-40.

Yang FJ et al. Green tea polyphenols block endotoxin-induced tumor necrosis factor alpha production and lethality in murine model. J Nutr 1998; 128:2334-40.

Yang TT, Koo MW. Hypocholesterolemic effects of Chinese tea. Pharmacol Res 1997; 35:505-12.

Yokogoshi H et al. Effect of theanine, r-glutamylethylamide, on brain monoamines and striatal dopamine release in conscious rats. Neurochem Res 1998; 23:667-73.

Yokozawa T et al. In vivo and in vitro studies on the radical scavenging activity of tea. J Agric Food Chem 1998; 46:2143-50.

You S. Study on feasibility of Chinese green tea polyphenols for preventing dental caries. Chung Hua Kou Chiang 1993; 28:197-9.

Zhang J, Kashket S. Inhibition of salivary amylase by black and green teas and their effects on the intraoral hydrolysis of starch. Caries Res 1998: 32:233-38.

Zhao BL et al. Scavenging effect of extracts of green tea and natural antioxidants on active oxygen species. Cell Biophys 1989; 14:175-85.


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